Are you IMMUNE to EBOLA or AIDS? You might be.

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You could be …

About 10% of white people are Immune to AIDS

C-C chemokine receptor type 5, also known as CCR5 or CD195, is a protein on the surface of white blood cells that is involved in the immune system as it acts as a receptor for chemokines. This is the process by which T cells are attracted to specific tissue and organ targets. Many forms of HIV, the virus that causes AIDS, initially use CCR5 to enter and infect host cells. A few individuals carry a mutation known as CCR5-Δ32 in the CCR5 gene, protecting them against these strains of HIV.

While CCR5 has multiple variants in its coding region, the deletion of a 32-bp segment results in a nonfunctional receptor, thus preventing HIV R5 entry; two copies of this allele provide strong protection against HIV infection. This allele is found in 5–14% of Europeans but is rare in Africans and Asians.
Now many scientists believe Black Plague was actually EBOLA.
Controversial new research suggests that contrary to the history books, the “Black Death” that devastated medieval Europe was not the bubonic plague, but rather an Ebola-like virus.
Medieval descriptions of the Black Death sound like the hemorrhagic fever caused by an Ebola-like virus, the authors say. Such fever strikes fast and causes blood vessels to burst underneath the skin, bringing out welts, similar to what British medical texts from the Middle Ages describe as “God’s tokens.”
The liquidization of internal organs that causes excruciating pain in Ebola victims matches the descriptions of historical autopsies on plague victims, which similarly describe internal organs being dissolved along with the appearance of a black liquid, according to the authors.
So your ancestors have probably come in contact with this virus before, and those that survived it, went on to breed.
Apocalyptic, mysterious plague killed millions of Native Americans.
Something horrific was happening in the Highlands of Mexico during that era, however. In 1545, almost immediately after the arrival in Mexico of a handful of de Soto Expedition survivors, a mysterious plague appeared in the Central Highlands of Mexico. After it settled in a Native community, between 85% and 100% of the population would quickly die. It was not a disease that Europeans recognized. In fact, very few, if any, Europeans even caught the disease.
Symptoms of the plague included high fevers, headache, and bleeding from the nose, ears, and mouth, accompanied by jaundice, severe abdominal and thoracic pain as well as acute neurological manifestations. Victims could feel perfectly healthy at breakfast, but be dead by supper.
At best victims might survive for 2-4 days. In the 1545 outbreak of this plague, 85% of the Native Americans died in the Central Highlands of Mexico, while 45% of the total Native population of Mexico died. In that era, the territory of Mexico included all of Central America and what is now the southwestern United States.
Epidemiologists currently believe that this killer disease was a strain of hantavirus, carried by certain wild rodents that somehow mutated into a hemorrhagic fever with symptoms similar to the Ebola Fever of Africa. To this day no one knows why this indigenous microbe suddenly became so deadly, or even how it was transmitted from village to village. No one knows either what caused the disease to seemingly disappear in the early 1800s.
De Soto’s men were allowed to dig up the graves of the deceased in that town to gather pearls buried as grave offerings. Undoubtedly, the pearls were contaminated with the microbe that caused the plague. The Spaniards carried those pearls as far as southwesternn Alabama before losing them during a battle. (Yet NONE of them died or even got sick.)
This video explains the way in which some white people are immune. How the virus fails to be able to enter their cells.

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Here is a doco about the discovery of Delta 32 gene.
A bit long but worth the watch.

Finally found it.
Yes it is a mutant gene which goes hand and hand with RH negative blood.
“prevalence of HIV serotypes is significantly higher (97.8%) among Rhesus D [positive] subjects than their Rhesus D [negative] counterparts in which 2.2% was recorded. This finding was in consonance with the results of Omoriegie et al. which reported that Rhesus D [positive] subjects were more susceptible to infections.”
Please Note ~ Rhesus D Positive means Rh+ and Rhesus D Negative means Rh-.
Hold the phone I have found it !!!
Overall, the prevalence of blood group O+ was higher than in the general population with highest rate of 62.9% in HIV+ pregnant women followed by 58.4% in HIV- pregnant women and 58.0% in non-pregnant women. No difference was observed in groups A+, B+, AB+, O- for the three categories of subjects studied. Blood groups B-, AB- were conspicuously absent in HIV+ pregnant women but non-significant in HIV pregnant women and the control. Type A- was very few in all the categories.
So it is not O negative it is B- or AB-. B is the significant blood type people who are B negative, can’t get AIDS.
more …
Overall Rh Positive (Rh+) for HIV+ pregnant women was 96.84%; for HIV- pregnant Women, it was 96.06% and for non-pregnant women-97.33%
while Rh-Negative Factor for HIV+ pregnant women was 3.16%; for HIV- group – 3.46% and non-pregnant women– 2.67%.
In the combined test, blood group O+ was the most numerous (62.9% for HIV+; 58.4% for HIV-; 58.0% for non-pregnant),
followed by A+ (19.4% for HIV+; 22.5% for HIV-; 22.3% for non-pregnant women), and B+ (13.4% for HIV+; 14.0% for HIV-; 16.0% for non-pregnant)
while O-, AB+ B-, A- were few for HIV- and AB- and B- were not found among HIV+ pregnant women.


4 thoughts on “Are you IMMUNE to EBOLA or AIDS? You might be.

  1. Delta 32 mutation has nothing to do with blood types period. It has to do with the mutation of CCR5 Delta 32 causing the white blood cells to have a protein barrier around the cells thus preventing certain virus to penetrate it and for a lack of a better word hitch a ride in the cell and travel to the lymph nodes were it disables the immune system. Scientist have done genetic testing in many countries and they found that Iceland, Azores and northern England have the highest number of people with CCR5 Delta 32 mutation. The best way to get correct information is to read science and health journals which is published by actual specialist in the field of human genome/genetics studies.

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